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Clinics is a gold open access electronic international journal that publishes peer-reviewed research in continuous flow of interest to clinicians and researchers in medical and biomedical sciences based on its originality, importance, conclusions and interest to readers.
The journal is committed to the principles of ethics, respect for the individual, humanization, honesty, pioneering spirit and excellence. The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. SRJ is a prestige metric based on the idea that not all citations are the same. SJR uses a similar algorithm as the Google page rank; it provides a quantitative and qualitative measure of the journal's impact.
SNIP measures contextual citation impact by wighting citations based on the total number of citations in a subject field. To evaluate the frequencies of iNKT cells and their subsets in patients with deep endometriosis.. A case-control study was conducted between and , with 73 patients distributed into two groups: 47 women with a histological diagnosis of endometriosis and 26 controls. Peripheral blood, endometriosis lesions, and healthy peritoneal samples were collected on the day of surgery to determine the frequencies of iNKT cells and subtypes via flow cytometry analysis..
The decreased number of total iNKT and DN iNKT cells in patients with endometriosis suggests that iNKT cells play a role in the pathogenesis of endometriosis and can be used to develop new diagnostic and therapeutic agents..
These clinical manifestations are heterogeneous and not always compatible with the severity or stage of the disease. Several studies have demonstrated the importance of the immune system in the pathogenesis of endometriosis. Disturbances in immunological homeostasis can facilitate implantation, proliferation, and angiogenesis of endometrial tissue in the peritoneum. In recent years, the importance of invariant Natural Killer T iNKT cells in the control of Th1, Th2, and Th17 immune responses and their relation to certain diseases has been demonstrated.